The first time I met my wife, we had an evening in the dining room of her house in New York.
We sat down and she brought out a box of tissues, a little glass bowl of orange juice, and a small box of cookies, which she offered to me to try.
I took a bite.
It was delicious.
But as the cookies slid down my throat, the chocolate chip cookies fell apart.
They had been sitting there all evening, and I’d never tasted them before.
And the rest of the chocolate chips?
They were still there.
I’d tasted them just as they had been the night before.
They were the very same chocolate chip cookie, the very very same cookie, and the very last one I’d eaten in my life.
I had never had cancer.
I’ve had several other conditions that have caused my pancreas to malfunction.
I have a rare, incurable disease called cancer.
When my doctor and I found out that I had a rare disease called breast cancer, we went to a local clinic to see a specialist who specializes in treating the disease.
The specialist suggested we visit a private facility in the suburbs to see how my disease was treating my body.
The treatment they offered was simple.
They took a sample of my breast tissue and gave it to a specialist to analyze for the rare disease.
But that was not the end of the treatment.
The specialists also did genetic testing to see if my cells had mutated.
After the genetic testing, they were able to find the mutations in my cells and see what they did to them.
That’s when I began to feel better.
The genetic testing revealed that my cancer had a mutation that made it more likely that it would metastasize.
In other words, my breast cancer cells were more likely to get into my lungs, into my blood vessels, and into my brain.
It seemed that the mutation in my cancer cells had been preventing them from reaching their destination.
In fact, this mutation made the cancer cells more resistant to being killed by other types of cancer drugs.
The mutation in the cancer was called BRCA1, which is a gene that helps make certain cells, called B cells, to grow.
And this mutation had been blocking the B cells from growing and multiplying.
So what could B cells do?
They could divide and multiply.
They could repair themselves.
They would form a network of cells called the myeloid lineage.
The myeloids are made of stem cells, the cells that are the first line of defense for your body.
Myeloid cells are found in every part of your body, from your bone marrow to your liver to your brain.
The stem cells were part of the cancer cell’s survival mechanism, and myelium, the stem cells that make up myelia, were responsible for the cells’ growth.
Myelin was created in myelin-rich skin cells that were found in the skin of my mother and my sister.
My myel cells were able at that time to differentiate into myeloma cells, and when they did, they could take on certain genetic information from the B cell that they had inherited from their mother.
Mymyelium was also part of a genetic pathway that could trigger the cancer to start multiplying.
This genetic information allowed the cancerous cells to multiply, but they were also able to repair themselves in response to new threats.
These cells were called mitotic cells.
In this way, the mitotic cell was able to keep the cancer growing and grow larger.
It also protected my mother from being harmed by the cancer.
At this point, my doctors and I were hopeful.
They wanted to test me for the BRCAs1 mutation.
They told me that if they could find it in my DNA, it would give them a clue as to how the B-cell mutations were affecting my body, so they could try and find a cure.
It turned out that the B+ gene in my B cells had a genetic mutation that gave it a boost of energy, which made it easier for the mitosis to repair itself.
But this boost of activity was only temporary.
In about 10 to 15 years, my body would be able to process all the excess energy it had been using, and then it would be hard to stop it.
And it was this resistance that would eventually lead to my cancer metastasizing.
When cancer is growing in a patient, the patient can’t control it, and so they can’t get any treatment that will stop it from spreading to other parts of their body.
They’re called metastatic.
And there are many ways that cancer cells can grow in a person’s body.
This is why the disease often develops in the lungs.
The lungs contain a population of B cells that keep them alive, and they also contain a large number of myelian cells, which are part of myelin that helps protect myelin from being damaged by